1832. The first mention of the cloth trade for which Kidderminster was

formerly noted occurs in 1334, when it was enacted that no one should make woollen cloth in the borough without the bailiff's seal. At the end of the 18th century the trade was still important, but it began to decline after the invention of machinery, probably owing to the poverty of the manufacturers. The manufacture of woollen goods was however replaced by that of carpets, introduced in 1735. At first only the "Kidderminster" carpets were made, but in 1749 a Brussels loom was set up in the town and Brussels carpets were soon produced in large quantities. See _Victoria County History: Worcestershire_; J. R. Burton, _A History of Kidderminster, with Short Accounts of some Neighbouring Parishes_ (1890). KIDNAPPING (from _kid_, a slang term for a child, and _nap_ or _nab_, to steal), originally the stealing and carrying away of children and others to serve as servants or labourers in the American plantations; it was defined by Blackstone as the forcible abduction or stealing away of a man, woman or child from their own country and sending them into another. The difference between kidnapping, abduction (q.v.) and false imprisonment is not very great; indeed, kidnapping may be said to be a form of assault and false imprisonment, aggravated by the carrying of the person to some other place. The term is, however, more commonly applied in England to the offence of taking away children from the possession of their parents. By the Offences against the Person Act 1861, "whosoever shall unlawfully, by force or fraud, lead or take away or decoy or entice away or detain any child under the age of fourteen years with intent to deprive any parent, guardian or other person having the lawful care or charge of such child of the possession of such child, or with intent to steal any article upon or about the person of such child, to whomsoever such article may belong, and whosoever shall with any such intent receive or harbour any such child, &c.," shall be guilty of felony, and is liable to penal servitude for not more than seven years, or to imprisonment for any term not more than two years with or without hard labour. The abduction or unlawfully taking away an unmarried girl under sixteen out of the possession and against the will of her father or mother, or any other person having the lawful care or charge of her, is a misdemeanour under the same act. The term is used in much the same sense in the United States. The kidnapping or forcible taking away of persons to serve at sea is treated under IMPRESSMENT. KIDNEY DISEASES.[1] (For the anatomy of the kidneys, see URINARY SYSTEM.) The results of morbid processes in the kidney may be grouped under three heads: the actual lesions produced, the effects of these on the composition of the urine, and the effects of the kidney-lesion on the body at large. Affections of the kidney are congenital or acquired. When acquired they may be the result of a pathological process limited to the kidney, in which case they are spoken of as primary, or an accompaniment of disease in other parts of the body, when they may be spoken of as secondary. _Congenital Affections._--The principal congenital affections are anomalies in the number or position of the kidneys or of their ducts; atrophy; cystic disease and growths. The most common abnormality is the existence of a _single kidney_; rarely a supernumerary kidney may be present. The presence of a single kidney may be due to failure of development, or to atrophy in foetal life; it may also be dependent on the fusion of originally separate kidneys in such a way as to lead to the formation of a _horse-shoe kidney_, the two organs being connected at their lower ends. In some cases of horse-shoe kidney the organs are united merely by fibrous tissue. Occasionally the two kidneys are fused end to end, with two ureters. A third variety is that where the fusion is more complete, producing a disk-like mass with two ureters. The kidneys may be situated in _abnormal positions_; thus they may be in front of the sacro-iliac articulation, in the pelvis, or in the iliac fossa. The importance of such displacements lies in the fact that the organs may be mistaken for tumours. In some cases _atrophy_ is associated with mal-development, so that only the medullary portion of the kidney is developed; in others it is associated with arterial obstruction, and sometimes it may be dependent upon obstruction of the ureter. In _congenital cystic disease_ the organ is transformed into a mass of cysts, and the enlargement of the kidneys may be so great as to produce difficulties in birth. The cystic degeneration is caused by obstruction of the uriniferous tubules or by anomalies in development, with persistence of portions of the Wolffian body. In some cases cystic degeneration is accompanied by anomalies in the ureters and in the arterial supply. _Growths of the kidney_ are sometimes found in infants; they are usually malignant, and may consist of a peculiar form of sarcoma, which has been spoken of as rhabdo-sarcoma, owing to the presence in the mass of involuntary muscular fibres. The existence of these tumours is dependent on anomalies of development; the tissue which forms the primitive kidney belongs to the same layer as that which gives rise to the muscular system (mesoblast). _Anomalies of the excretory ducts_: in some cases the ureter is double, in others it is greatly dilated; in others the pelvis of the kidney may be greatly dilated, with or without dilatation of the ureter. _Acquired Affections. Movable Kidney._--One or both of the kidneys in the adult may be preternaturally mobile. This condition is more common in women, and is usually the result of a severe shaking or other form of injury, or of the abdominal walls becoming lax as a sequel to abdominal distension, to emaciation or pregnancy, or to the effects of tight-lacing. The more extreme forms of movable kidney are dependent, generally, on anomalies in the arrangement of the peritoneum, so that the organ has a partial mesentery; and to this condition, where the kidney can be moved freely from one part of the abdomen to another, the term _floating kidney_ is applied. But more usually the organ is loose under the peritoneum, and not efficiently supported in its fatty bed. Movable kidney produces a variety of symptoms, such as pain in the loin and back, faintness, nausea and vomiting--and the function of the organ may be seriously interfered with, owing to the ureter becoming kinked. In this way hydronephrosis, or distension of the kidney with urine, may be produced. The return of blood through the renal vein may also be hindered, and temporary vascular engorgement of the kidney, with haematuria, may be produced. In some cases the movable kidney may be satisfactorily kept in its place by a pad and belt, but in other cases an operation has to be undertaken. This consists in exposing the kidney (generally the right) through an incision below the last rib, and fixing it in its proper position by several permanent sutures of silk or silkworm gut. The operation is neither difficult nor dangerous, and its results are excellent. _Embolism._--The arrangement of the blood-vessels of the kidney is peculiarly favourable to the production of wedge-shaped areas of necrosis, the result of a blocking by clots. Sometimes the clot is detached from the interior of the heart, the effect being an arrest of the circulation in the part of the kidney supplied by the blocked artery. In other cases, the plug is infective owing to the presence of septic micro-organisms, and this is likely to lead to the formation of small pyaemic abscesses. It is exceptional for the large branches of the renal artery to be blocked, so that the symptoms produced in the ordinary cases are only the temporary appearance of blood or albumen in the urine. Blocking of the main renal vessels as a result of disease of the walls of the vessels may lead to disorganization of the kidneys. Blocking of the veins, leading to extreme congestion of the kidney, also occurs. It is seen in cases of extreme weakness and wasting, sometimes in septic conditions, as in puerperal pyaemia, where a clot, formed first in one of the pelvic veins, may spread up the vena cava and secondarily block the renal veins. Thrombosis of the renal vein also occurs in malignant disease of the kidney and in certain forms of chronic Bright's disease. _Passive congestion_ of the kidneys occurs in heart-diseases and lung-diseases, where the return of venous blood is interfered with. It may also be produced by tumours pressing on the vena cava. The engorged kidneys become brownish red, enlarged and fibroid, and they secrete a scanty, high-coloured urine. _Active congestion_ is produced by the excretion in the urine of such materials as turpentine and cantharides and the toxins of various diseases. These irritants produce engorgement and inflammation of the kidney, much as they would that of any other structures with which they come in contact. Renal disturbance is often the result of the excretion of microbic poisons. Extreme congestion of the kidneys may be produced by exposure to cold, owing to some intimate relationship existing between the cutaneous and the renal vessels, the constriction of the one being accompanied by the dilatation of the other. Infective diseases, such as typhoid fever, pneumonia, scarlet fever, in fact, most acute specific diseases, produce during their height a temporary nephritis, not usually followed by permanent alteration in the kidney; but some acute diseases cause a nephritis which may lay the foundation of permanent renal disease. This is most common as a result of scarlet fever. _Bright's disease_ is the term applied to certain varieties of acute and chronic inflammation of the kidney. Three forms are usually recognized--acute, chronic and the granular or cirrhotic kidney. In the more common form of granular kidney the renal lesion is only part of a widespread affection involving the whole arterial system, and is not actually related to Bright's disease. _Chronic Bright's disease_ is sometimes the sequel to acute Bright's disease, but in a great number of cases the malady is chronic from the beginning. The lesions of the kidney are probably produced by irritation of the kidney-structures owing to the excretion of toxic substances either ingested or formed in the body; it is thought by some that the malady may arise as a result of exposure to cold. The principal causes of Bright's disease are alcoholism, gout, pregnancy and the action of such poisons as lead; it may also occur as a sequel to acute diseases, such as scarlet fever. Persons following certain occupations are peculiarly liable to Bright's disease, e.g. engineers who work in hot shops and pass out into the cold air scantily clothed; and painters, in whom the malady is dependent on the action of lead on the kidney. In the case of alcohol and lead the poison is ingested; in the case of scarlet fever, pneumonia, and perhaps pregnancy, the toxic agent causing the renal affection is formed in the body. In Bright's disease all the elements of the kidney, the glomeruli, the tubular epithelium, and the interstitial tissue, are affected. When the disease follows scarlet fever, the glomerular structures are mostly affected, the capsules being thickened by fibrous tissue, and the glomerular tuft compressed and atrophied. The epithelium of the convoluted tubules undergoes degeneration; considerable quantities of it are shed, and form the well-known casts in the urine. The tubules become blocked by the epithelium, and distended with the pent-up urine; this is one cause of the increase in size that the kidneys undergo in certain forms of Bright's disease. The lesions in the tubules and in the glomeruli are not generally uniform. The interstitial tissue is always affected, and exudation, proliferation and formation of fibrous tissue occur. In the granular and contracted kidney the lesion in the interstitial tissue reaches a high degree of development, little renal secreting tissue being left. Such tubules as remain are dilated, and the epithelium lining them is altered, the cells becoming hyaline and losing their structure. The vessels are narrowed owing to thickening of the subendothelial layer, and the muscular coat undergoes hypertrophic and fibroid changes, so that the vessels are abnormally rigid. When the overgrowth of fibrous tissue is considerable, the surface of the organ becomes uneven, and it is for this reason that the term _granular kidney_ has been applied to the condition. In acute Bright's disease the kidney is increased in size and engorged with blood, the changes described above being in active progress. In the chronic form the kidney may be large or small, and is usually white or mottled. If large, the cortex is thickened, pale and waxy, and the pyramids are congested; if small, the fibrous change has advanced and the cortex is diminished. Bright's disease, both acute and chronic, is essentially a disease of the cortical secreting portion of the kidney. The true granular kidney, classified by some as a third variety, is usually part of a general arterial degeneration, the overgrowth of fibrous tissue in the kidney and the lesions in the arteries being well marked. The principal degenerations affecting the kidney are the fatty and the albuminoid. _Fatty degeneration_ often reaches a high degree in alcoholics, where fatty degeneration of the heart and liver are also present. _Albuminoid disease_ is frequently associated with some varieties of Bright's disease, and is also seen as a result of chronic bone disease, or of long-continued suppuration involving other parts of the body, or of syphilis. It is due to irritation of the kidneys by toxic products. _Growths of the Kidney._--The principal growths are tubercle, adenoma, sarcoma and carcinoma. In addition, fatty and fibrous growths, the nodules of glanders and the gummata of syphilis, may be mentioned. Tuberculous disease is sometimes primary; more frequently it is secondary to tubercle in other portions of the genito-urinary apparatus. The genito-urinary tract may be infected by tubercle in two ways; _ascending_, in which the primary lesion is in the testicle, epididymis, or urinary bladder, the lesion travelling up by the ureter or the lymphatics to the kidney; _descending_, where the tubercle bacillus reaches the kidney through the blood-vessels. In the latter case, miliary tubercles, as scattered granules, are seen, especially in the cortex of the kidney; the lesion is likely to be bilateral. In primary tuberculosis, and in ascending tuberculosis, the lesion is at first unilateral. _Malignant disease_ of the kidney takes the form of sarcoma or carcinoma. Sometimes it is dependent on the malignant growths starting in what are spoken of as "adrenal rests" in the cortex of the kidney. Sarcoma is most often seen in the young; carcinoma in the middle-aged and elderly. Carcinoma may be primary or secondary, but the kidney is not so prone to malignant disease as other organs, such as the stomach, bowel or liver. _Cystic Kidneys._--Cysts may be single--sometimes of large size. Scattered small cysts are met with in chronic Bright's disease and in granular contracted kidney, where the dilatation of tubules reaches a high degree. Certain growths, such as adenomata, are liable to cystic degeneration, and cysts are also found in malignant disease. Finally, there is a rare condition of general cystic disease somewhat similar to the congenital affection. In this form the kidneys, greatly enlarged, consist of a congeries of cysts separated by the remains of renal tissue. _Parasitic Affections._--The more common parasites affecting the kidney, or some other portion of the urinary tract, and causing disease, are filaria, bilharzia and the cysticercus form of the _taenia echinococcus_ (hydatids). The presence of _filaria_ in the thoracic duct and other lymph-channels may determine the presence of chyle in the urine, together with the ova and young forms of the filaria, owing to the distension and rupture of a lymphatic vessel into some portion of the urinary tract. This is the common cause of chyluria in hot climates, but chyluria is occasionally seen in the United Kingdom without filaria. _Bilharzia_, especially in Egypt and South Africa, causes haematuria. The cysticercus form of the _taenia echinococcus_ leads to the production of hydatid cysts in the kidney; this organ, however, is not so often affected as the liver. _Stone in the Kidney._--Calculi are frequently found in the kidney, consisting usually of uric acid, sometimes of oxalates, more rarely of phosphates. Calculous disease of the bladder (q.v.) is generally the sequel to the formation of a stone in the kidney, which, passing down, becomes coated by the salts in the urine. Calculi are usually formed in the pelvis of the kidney, and their formation is dependent either on the excessive amounts of uric acid, oxalic acid, &c., in the urine, or on an alteration in the composition of the urine, such as increased acidity, or on uric acid or oxalate of lime being present in an abnormal amount. The formation of abnormal crystals is often due to the presence of some colloid, such as blood, mucus or albumen, in the secretion, modifying the crystalline form. Once a minute calculus has been formed, its subsequent growth is highly probable, owing to the deposition on it of the urinary constituent forming it. Calculi formed in the pelvis of the kidney may be single and may reach a very large size, forming, indeed, an actual cast of the interior of the expanded kidney. At other times they are multiple and of varying size. They may give rise to no symptoms, or on the other hand may cause distressing renal colic, especially when they are small and loose and are passed or are trying to be passed. Serious complications may result from the presence of a stone in the kidney, such as hydronephrosis, from the urinary secretion being pent up behind the obstruction, or complete suppression, which is apparently produced reflexly through the nervous system. In such cases the surgical removal of the stone is often followed by the restoration of the renal secretion. The symptoms of _renal calculus_ may be very slight, or they may be entirely absent if the stone is moulding itself into the interior of the kidney; but if the stone is movable, heavy and rough, it may cause great distress, especially during exercise. There will probably be blood in the urine; and there will be pain in the loin and thigh and down into the testicle. The testicle also may be drawn up by its suspensory muscle, and there may be irritability of the bladder. With stone in one kidney the pains may be actually referred to the kidney of the other side. Generally, but not always, there is tenderness in the loin. If the stone is composed of lime it may throw a shadow on the Röntgen plate, but other stones may give no shadow. _Renal colic_ is the acute pain felt when a small stone is travelling down the ureter to the bladder. The pain is at times so acute that fomentations, morphia and hot baths fail to ease it, and nothing short of chloroform gives relief. For the _operative treatment of renal calculus_ an incision is made a little below the last rib, and, the muscles having been traversed, the kidney is reached on the surface which is not covered by peritoneum. Most likely the stone is then felt, so it is cut down upon and removed. If it is not discoverable on gently pinching the kidney between the finger and thumb, the kidney had better be opened in its convex border and explored by the finger. Often it has happened that when a man has presented most of the symptoms of renal calculus and has been operated on with a negative result as regards finding a stone, all the symptoms have nevertheless disappeared as the direct result of the blank operation. _Pyelitis._--Inflammation of the pelvis of the kidney is generally produced by the extension of gonorrhoeal or other septic inflammation upwards from the bladder and lower urinary tract, or by the presence of stone or of tubercle in the pelvis of the kidney. Pyonephrosis, or distension of the kidney with pus, may result as a sequel to pyelitis or as a complication of hydronephrosis; in many cases the inflammation spreads to the capsule of the kidney, and leads to the formation of an abscess outside the kidney--a _perinephritic abscess_. In some cases a perinephritic abscess results from a septic plug in a blood-vessel of the kidney, or it may occur as the result of an injury to the loose cellular tissue surrounding the kidney, without lesion of the kidney. _Hydronephrosis_, or distension of the kidney with pent-up urine, results from obstruction of the ureter, although all obstructions of the ureter are not followed by it, calculous obstruction, as already noted, often causing complete suppression of urine. Obstruction of the ureter, causing hydronephrosis, is likely to be due to the impaction of a stone, or to pressure on the ureter from a tumour in the pelvis--as, for instance, a cancer of the uterus--or to some abnormality of the ureter. Sometimes a kink of the ureter of a movable kidney causes hydronephrosis. The hydronephrosis produced by obstruction of the ureter may be intermittent; and when a certain degree of distension is produced, either as a result of the shifting of the calculus or of some other cause, the obstruction is temporarily relieved in a great outflow of urine, and the urinary discharge is re-established. When the hydronephrosis has long existed the kidney is converted into a sac, the remains of the renal tissues being spread out as a thin layer. _Effects on the Urine._--Diseases of the kidney produce alterations in the composition of the urine; either the proportion of the normal constituents being altered, or substances not normally present being excreted. In most diseases the quantity of urinary water is diminished, especially in those in which the activity of the circulation is impaired. There are diseases, however, more especially the granular kidney and certain forms of chronic Bright's disease, in which the quantity of urinary water is considerably increased, notwithstanding the profound anatomical changes that have occurred in the kidney. There are two forms of suppression of the urine: one is _obstructive suppression_, seen where the ureter is blocked by stone or other morbid process; the other is _non-obstructive suppression_, which is apt to occur in advanced diseases of the kidney. In other cases complete suppression may occur as the result of injuries to distant parts of the body, as after severe surgical operations. In some diseases in which the quantity of urinary water excreted is normal, or even greater than normal, the efficiency of the renal activity is really diminished, inasmuch as the urine contains few solids. In estimating the efficiency of the kidneys, it is necessary to take into consideration the so-called "solid urine," that is to say, the quantity of solid matter daily excreted, as shown by the specific gravity of the urine. The nitrogenous constituents--urea, uric acid, creatinin, &c.--vary greatly in amount in different diseases. In most renal diseases the quantities of these substances are diminished because of the physiological impairment of the kidney. The chief abnormal constituents of the urine are serum-albumen, serum-globulin, albumoses (albuminuria), blood (haematuria), blood pigment (haemoglobinuria), pus (pyuria), chyle (chyluria) and pigments such as melanuria and urobilinuria. _Effects on the Body at large._--These may be divided into the persistent and the intermittent or transitory. The most important persistent effects produced by disease of the kidney are, first, nutritional changes leading to general ill health, wasting and cachexia; and, secondly, certain cardio-vascular phenomena, such as enlargement (hypertrophy) of the heart, and thickening of the inner, and degeneration of the middle, coat of the smaller arteries. Amongst the intermittent or transitory effects are dropsy, secondary inflammations of certain organs and serous cavities, and uraemia. Some of these effects are seen in every form of severe kidney disease, and uraemia may occur in any advanced kidney disease. Renal dropsy is chiefly seen in certain forms of Bright's disease, and the cardiac and arterial changes are commonest in cases of granular or contracted kidney, but may be absent in other diseases which destroy the kidney tissue, such as hydronephrosis. _Uraemia_ is a toxic condition, and three varieties of it are recognized--the acute, the chronic and the latent. Many of these effects are dependent upon the action of poisons retained in the body owing to the deficient action of the kidneys. It is also probable that abnormal substances having a toxic action are produced as a result of a perverted metabolism. Uraemia is of toxic origin, and it is probable that the dropsy of renal disease is due to effects produced in the capillaries by the presence of abnormal substances in the blood. High arterial tension, cardiac hypertrophy and arterial degeneration may also be of toxic origin, or they may be produced by an attempt of the body to maintain an active circulation through the greatly diminished amount of kidney tissue available. _Rupture of the kidney_ may result from a kick or other direct injury. Vomiting and collapse are likely to ensue, and most likely blood will appear in the urine, or a tumour composed of blood and urine may form in the renal region. An incision made into the swelling from the loin may enable the surgeon to see the torn kidney. An attempt should be made to save the kidney by suturing and draining; unless the damage is obviously past repair, the kidney should not be removed without giving nature a chance. (J. R. B.; E. O.*) FOOTNOTE: [1] The word "kidney" first appears in the early part of the 14th century in the form _kidenei_, with plural _kideneiren_, _kideneris_, _kidneers_, &c. It has been assumed that the second part of the word is "neer" or "near" (cf. Ger. _Niere_), the common dialect word for "kidney" in northern, north midland and eastern counties of England (see J. Wright, _English Dialect Dictionary_, 1903, _s.v._ Near), and that the first part represents the O.E. _cwið_, belly, womb; this the _New English Dictionary_ considers improbable; there is only one doubtful instance of singular _kidnere_ and the ordinary form ended in -_ei_ or _ey_. Possibly this represents M.E. _ey_, plur. _eyren_, egg, the name being given from the resemblance in shape. The first part is uncertain. KIDWELLY (_Cydweli_), a decayed market-town and municipal borough of Carmarthenshire, Wales, situated (as its name implies) near the junction of two streams, the Gwendraeth Fawr and the Gwendraeth Fach, a short distance from the shores of Carmarthen Bay. Pop. (1901), 2285. It has a station on the Great Western railway. The chief attraction of Kidwelly is its magnificent and well-preserved castle, one of the finest in South Wales, dating chiefly from the 13th century and admirably situated on a knoll above the Gwendraeth Fach. The parish church of St Mary, of the 14th century, possesses a lofty tower with a spire. The quiet little town has had a stirring history. It was a place of some importance when William de Londres, a companion of Fitz Hamon and his conquering knights, first erected a castle here. In 1135 Kidwelly was furiously attacked by Gwenllian, wife of Griffith ap Rhys, prince of South Wales, and a battle, fought close to the town at a place still known as Maes Gwenllian, ended in the total defeat and subsequent execution of the Welsh princess. Later, the extensive lordship of Kidwelly became the property through marriage of Henry, earl of Lancaster, and to this circumstance is due the exclusive jurisdiction of the town. Kidwelly received its first charter of incorporation from Henry VI.; its present charter dating from 1618. The decline of Kidwelly is due to the accumulation of sand at the mouth of the river, and to the consequent prosperity of the neighbouring Llanelly. KIEF, KEF or KEIF (a colloquial form of the Arabic _kaif_, pleasure or enjoyment), the state of drowsy contentment produced by the use of narcotics. To "do kef," or to "make kef," is to pass the time in such a state. The word is used in northern Africa, especially in Morocco, for the drug used for the purpose. KIEL, the chief naval port of Germany on the Baltic, a town of the Prussian province of Schleswig-Holstein. Pop. (1900), 107,938; (1905), 163,710, including the incorporated suburbs. It is beautifully situated at the southern end on the Kieler Busen (bay or harbour of Kiel), 70 m. by rail N. from Hamburg. It consists of a somewhat cramped old town, lying between the harbour and a sheet of water called Kleiner Kiel, and a better built and more spacious new town, which has been increased by the incorporation of the garden suburbs of Brunswick and Düsternbrook. In the old town stands the palace, built in the 13th century, enlarged in the 18th and restored after a fire in 1838. It was once the seat of the dukes of Holstein-Gottorp, who resided here from 1721 to 1773, and became the residence of Prince Henry of Prussia. Other buildings are the church of St Nicholas (restored in 1877-1884), dating from 1240, with a lofty steeple; the old town-hall on the market square; the church of the Holy Ghost; three fine modern churches, those of St James, and St Jürgen and of St Ansgar; and the theatre. Further to the north and facing the bay is the university, founded in 1665 by Christian Albert, duke of Schleswig, and named after him "Christian Albertina." The new buildings were erected in 1876, and connected with them are a library of 240,000 volumes, a zoological museum, a hospital, a botanical garden and a school of forestry. The university, which is celebrated as a medical school, is attended by nearly 1000 students, and has a teaching staff of over 100 professors and docents. Among other scientific and educational institutions are the Schleswig-Holstein museum of national antiquities in the old university buildings, the Thaulow museum (rich in Schleswig-Holstein wood-carving of the 16th and 17th centuries), the naval academy, the naval school and the school for engineers. The pride of Kiel is its magnificent harbour, which has a comparatively uniform depth of water, averaging 40 ft., and close to the shores 20 ft. Its length is 11 m. and its breadth varies from ¼ m. at the southern end to 4½ m. at the mouth. Its defences, which include two forts on the west and four on the east side, all situated about 5 m. from the head of the harbour at the place (Friedrichsort) where its shores approach one another, make it a place of great strategic strength. The imperial docks (five in all) and ship-building yards are on the east side facing the town, between Gaarden and Ellerbeck, and comprise basins capable of containing the largest war-ships afloat. The imperial yard employs 7000 hands, and another 7000 are employed in two large private ship-building works, the Germania (Krupp's) and Howalds'. The Kaiser Wilhelm Canal, commonly called the Kiel Canal, connecting the Baltic with the North Sea at Brunsbüttel, has its eastern entrance at Wik, 1½ m. N. of Kiel (see GERMANY: _Waterways_). The town and adjacent villages, e.g. Wik, Heikendorf and Laboe, are resorted to for sea-bathing, and in June of each year a regatta, attended by yachts from all countries, is held. The _Kieler Woche_ is one of the principal social events in Germany, and corresponds to the "Cowes week" in England. Kiel is connected by day and night services with Korsör in Denmark by express passenger boats. The harbour yields sprats which are in great repute. The principal industries are those connected with the imperial navy and ship-building, but embrace also flour-mills, oil-works, iron-foundries, printing-works, saw-mills, breweries, brick-works, soap-making and fish-curing. There is an important trade in coal, timber, cereals, fish, butter and cheese. The name of Kiel appears as early as the 10th century in the form Kyl (probably from the Anglo-Saxon _Kille_ = a safe place for ships). Kiel is mentioned as a city in the next century; in 1242 it received the Lübeck rights; in the 14th century it acquired various trading privileges, having in 1284 entered the Hanseatic League. In recent times Kiel has been associated with the peace concluded in January 1814 between Great Britain, Denmark and Sweden, by which Norway was ceded to Sweden. In 1773 Kiel became part of Denmark, and in 1866 it passed with the rest of Schleswig-Holstein to Prussia. Since being made a great naval arsenal, Kiel has rapidly developed in prosperity and population. See Prahl, _Chronika der Stadt Kiel_ (Kiel, 1856); Erichsen, _Topographie des Landkreises Kiel_ (Kiel, 1898); H. Eckardt, _Alt-Kiel in Wort und Bild_ (Kiel, 1899); P. Hasse, _Das Kieler Stadtbuch, 1264-1289_ (Kiel, 1875); _Das älteste Kieler Rentebuch 1300, 1487_, edited by C. Reuter (Kiel, 1893); _Das zweite Kieler Rentebuch 1487, 1586_, edited by W. Stern (Kiel, 1904); and the _Mitteilungen der Gesellschaft für Kieler Stadtgeschichte_ (Kiel, 1877, 1904). KIELCE, a government in the south-west of Russian Poland, surrounded by the governments of Piotrkow and Radom and by Austrian Galicia. Area, 3896 sq. m. Its surface is an elevated plateau 800 to 1000 ft. in altitude, intersected in the north-east by a range of hills reaching 1350 ft. and deeply trenched in the south. It is drained by the Vistula on its south-east border, and by its tributaries, the Nida and the Pilica, which have a very rapid fall and give rise to inundations. Silurian and Devonian quartzites, dolomite, limestones and sandstones prevail in the north, and contain rich iron ores, lead and copper ores. Carboniferous deposits containing rich coal seams occur chiefly in the south, and extend into the government of Piotrkow. Permian limestones and sandstones exist in the south. The Triassic deposits contain very rich zinc ores of considerable thickness and lead. The Jurassic deposits consist of iron-clays and limestones, containing large caves. The Cretaceous deposits yield gypsum, chalk and sulphur. White and black marble are also extracted. The soil is of great variety and fertile in parts, but owing to the proximity of the Carpathians, the climate is more severe than might be expected. Rye, wheat, oats, barley and buckwheat are grown; modern intensive culture is spreading, and land fetches high prices, the more so as the peasants' allotments were small at the outset and are steadily decreasing. Out of a total of 2,193,300 acres suitable for cultivation 53.4% are actually cultivated. Grain is exported. Gardening is a thriving industry in the south; beet is grown for sugar in the south-east. Industries are considerably developed: zinc ores are extracted, as well as some iron and a little sulphur. Tiles, metallic goods, leather, timber goods and flour are the chief products of the manufactures. Pop. (1897), 765,212, for the most part Poles, with 11% Jews; (1906, estimated), 910,900. By religion 88% of the people are Roman Catholics. Kielce is divided into seven districts, the chief towns of which, with populations in 1897, are Kielce (q.v.), Jedrzejow (Russ. Andreyev, 5010), Miechow (4156), Olkusz (3491), Pinczów (8095), Stopnica (4659) and Wloszczowa (23,065). KIELCE, a town of Russian Poland, capital of the above government, 152 m. by rail S. of Warsaw, situated in a picturesque hilly country. Pop. (1890), 12,775; (1897), 23,189. It has a castle, built in 1638 and for some time inhabited by Charles XII.; it was renowned for its portrait gallery and the library of Zaluski, which was taken to St Petersburg. The squares and boulevards are lined with handsome modern buildings. The principal factories are hemp-spinning, cotton-printing and cement works. The town was founded in 1173 by a bishop of Cracow. In the 16th century it was famous for its copper mines, but they are no longer worked. KIEPERT, HEINRICH (1818-1899), German geographer, was born at Berlin on the 31st of July 1818. He was educated at the university there, studying especially history, philology and geography. In 1840-1846, in collaboration with Karl Ritter, he issued his first work, _Atlas von Hellas und den hellenischen Kolonien_, which brought him at once into eminence in the sphere of ancient historical cartography. In 1848 his _Historisch-geographischer Atlas der alten Welt_ appeared, and in 1854 the first edition of the _Atlas antiquus_, which has obtained very wide recognition, being issued in English, French, Russian, Dutch and Italian. In 1894 Kiepert produced the first part of a larger atlas of the ancient world under the title _Formae orbis antiqui_; his valuable maps in _Corpus inscriptionum latinarum_ must also be mentioned. In 1877-1878 his _Lehrbuch der alten Geographie_ was published, and in 1879 _Leitfaden der alten Geographie_, which was translated into English (_A Manual of Ancient Geography_, 1881) and into French. Among Kiepert's general works one of the most important was the excellent _Neuer Handatlas über alle Teile der Erde_ (1855 et seq.), and he also compiled a large number of special and educational maps. Asia Minor was an area in which he took particular interest. He visited it four times in 1841-1888; and his first map (1843-1846), together with his _Karte des osmanischen Reiches in Asien_ (1844 and 1869), formed the highest authority for the geography of the region. Kiepert was professor of geography in the university of Berlin from 1854. He died at Berlin on the 21st of April 1899. He left unpublished considerable material in various departments of his work, and with the assistance of this his son Richard (b. 1846), who followed his father's career, was enabled to issue a map of Asia Minor in 24 sheets, on a scale of 1 : 400,000 (1902 et seq.), and to carry on the issue of _Formae orbis antiqui_. KIERKEGAARD, SÖREN AABY (1813-1855), Danish philosopher, the seventh child of a Jutland hosier, was born in Copenhagen on the 5th of May